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Interstitial fibrosis/uterine fibroids (IF/UF) is a common, progressive fibrosis disorder of the female genital tract. It is a major cause of infertility, especially in women of reproductive age. The exact pathogenesis of IF/UF has not been fully elucidated, but there are several mechanisms that play a key role in its development and progression. For the treatment of IF/UF, it is crucial to understand the pathogenesis of the disease and how to stop or slow the progression of IF/UF. A wide variety of therapeutic agents have been developed for IF/UF, including hormone therapies (gonadotropin-releasing hormone agonists, levonorgestrel-releasing intrauterine systems), anti-inflammatory agents (nonsteroidal anti-inflammatory drugs, cyclooxygenase-2 inhibitors), antifibrotic agents (tranilast), and anticancer drugs. Tranilast is a membrane-permeable, non-selective broad-spectrum inhibitor of protein kinase C (PKC). Treatment with tranilast can effectively block TGF-β1-induced EMT and relieve IF/UF development. Although the efficacy of tranilast is limited and most of patients are intolerant to the drug, tranilast is an attractive and relatively safe agent to treat IF/UF. In this review, we describe the background and current status of IF/UF, the pathogenesis and mechanisms of fibrosis, and some recent advances in the development of antifibrotic therapy.X-ray Studies
The first part of this data set is an analysis of
long-term X-ray flux from a supernova remnant
we have been looking at for the last two years,
the exploding star W44.
The second part is a reanalysis of the X-ray flux
from the Cygnus Loop region of the galaxy
Cygnus X-1.
Data for this plot were taken from the
NASA HEASARC X-Ray/Gamma-Ray Web page.
By viewing this online data set you are
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